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The principal focus of this study is to understand the mechanism for the association between macroprolactinomas and hyperprolactinaemia. As an adjunct to this study, the mechanisms involved in and the relationship between familial prolactin deficiency and hyperprolactinaemia in the absence of macroprolactinomas were also examined. This was a morphological study using 3H-thymidine labelling to demonstrate actively proliferating cells in macroprolactinomas in a 14-year-old woman with familial prolactin deficiency, hyperprolactinaemia, and macroprolactinomas. In addition, the changes in cell proliferation and involution of macroprolactinomas in this patient were examined by using conventional histology and a modified histological technique to demonstrate and quantify senile adenohypophysial cell involution. On the basis of the results
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