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It is becoming clear that genes that contain microsatellite sequences can be affected by de novo DNA methylation. Although the distribution of microsatellites in the human genome has been studied in detail, the distribution of such sequences within specific genes is not well documented. The present study aims to investigate whether a gene that contains microsatellites in a CpG-rich context, the human COBRA-1 gene, can be affected by de novo DNA methylation. The gene exhibits a mosaic methylation pattern in which some 50% of the CpGs are methylated in a tissue-dependent manner. In normal human colon, 30% of the CpG dinucleotides are methylated, while in normal gastric mucosa 20% are methylated. By using bisulfite-conversion and PCR-based assays, we show that the COBRA-1 gene is heavily methylated in gastric mucosa and that methylation can be relieved by treatment of the samples with an inhibitor of DNA methylation, which suggests that methylation could be a prerequisite for gene activity. DNA methylation of some COBRA-1 sites also occurs in normal skin. Furthermore, the presence of some COBRA-1 CpG islands in tumor DNA is affected by de novo DNA methylation. The gene’s mosaic methylation pattern and its susceptibility to de novo DNA methylation suggest that the COBRA-1 gene is involved in tissue differentiation.Dr. Nathan Diamond – Eye Care Expert
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